The pciniaresentation describe the life cycle of Pythium, Albugo, Erysiphae, Claviceps, Ustilago, Puccinia. both sexual spores (called oospores) and asexual spores (called sporangia) in a many-stage (polycyclic) disease cycle. infection stag heads oospore productt0n zoo spores. \. SEXUAL c germ1not1~. Oospores -overwintering spores. 8. Figure 8. Life cycle of Albugo candlda.

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Controlling white rust is very difficult due to the nature of the ‘Albugo’ pathogen.

Between the haustorium and the host plasma membrane is an encapsulation. Finally they settle down on the host, retract the flagella and albguo off.

This mode of development of sporangia is termed per-current proliferation. This papilla-like oogonial bulging is called the receptive papilla It is functionless. The oospore alongwith the fusion nucleus is called oospore Fig.


It contains about nuclei and dense cytoplasm. Albygo Wikipedia, the free encyclopedia. Symptoms of white rust caused by Albugo typically include yellow lesions on the upper leaf surface and white pustules on the underside of the leaf. The fungus mycelium grows vigorously. The rest of the nuclei disintegrate before fertilization. From then onwards hyphal growth increases rapidly.

albugo-life-cycle – Study Solutions

Each daughter protoplast shapes into a slightly concave-convex zoospore E. The place of meiosis in Albugo is still under dispute.


This is why identification of specific hosts crops and possible weeds is necessary to determine range and location of control methods.

This causes the leaf surface to bulge. The second sporangium is delimited in the same manner as the first. Septa remain suppressed in the actively growing hyphae but appear to separate reproductive structures and to seal off injured parts. It is surrounded a,bugo periplasm rich in vacuolate cytoplasm containing membranous organelles. After swimming for some time in water, they settle down on the host. So, the oospore is uninucleate. These are called biological forms or physiological forms.

The hyphae branch and ramify within the host attacking the tissues adjoining the point of infection.

Albugo: Habitat, Symptoms and Reproduction | Mastigomycotina

At the end of mitosis one daughter nucleus of each spindle goes to the ooplasm and the other to the periplasm F. Towards the end of sporangial maturation, the dictyosomes become quiescent, mitochondria decreased lifr number and also the amount of endoplasmic reticulum. According to StevensSansome and Sansomethe thallus of Albugo is diploid and the meiosis occurs in gametangla i. The antheridium and oogonium are formed near each other on hyphal branches.

Sometimes the lf is intercalary in position. Views Read Edit View history.

Fycle oospore produces forty to sixty zoospores. The terminal end of the sporangiophore becomes constricted and sporangium contains nuclei and cytoplasm. At the point of contact of antheridium with the oogonium, the walls become very thin.


According to one view, the sporangial chains in Albugo are abstricted by percurrent proliferation.

Albugo| Life cycle| White Rust| Peronosporales|

This pathogen can eliminate viable production of susceptible crops in specific fields indefinitely if infection is widespread over many years. Cycoe latter is then cut off by a cross wall from the rest of the male hypha B. On removing or displacing the attached antheridium Tewan and Skoropad observed a clear hole surrounded by some fibrous material. The asexual reproduction takes place by conidia, condiosporangia or zoosporangia. These zoospores then swim in a film of water to a suitable site and each one produces a germ tube – like that of the sporangium – that penetrates the stoma.

Soon the encysted zoospore cyst puts out a germ cyccle E which enters the host tissue through a stoma. White rust plant diseases caused by Albugo fungal-like pathogens should not be confused with White Pine Blister RustChrysanthemum white rust or any fungal rustsall of which are also plant diseases but have completely different symptoms and causal pathogens.

Favorable conditions for the dispersal and consequent infection of white rust from diseased to healthy plants are most common in the autumn and spring seasons.